Summarize the for patients with suspected resistance?
In inflammatory states, platelets or monocytes may express COX-2, which aspirin inhibits less effectively than COX-1. 3. Genetic Polymorphisms Summarize the for patients with suspected resistance
Simulates high-shear blood flow to measure the "closure time" of a microscopic aperture. It is sensitive but less specific for aspirin alone. platelets or monocytes may express COX-2
A non-invasive metabolic marker. High levels in urine suggest that aspirin is not successfully suppressing thromboxane production. 🏥 Clinical Implications Summarize the for patients with suspected resistance
Platelets can be activated via pathways not targeted by aspirin (e.g., ADP, thrombin, or collagen).